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YK 11 SARM - Dragon Pharma

Dragon Pharma

YK 11 SARM - Dragon Pharma

Oral · 10 mg/tab · 100 tabs

In stock · ships within 24h Out of stock Ships from International · U.S Domestic
CompoundYK-11 (myostatin SARM)
ClassSARM, steroidal
Half-lifeNot established
DetectionNot established
Liver toxicityClass risk, unquantified
Water retentionNone reported
One 10 mg tablet per day is the only fixed unit the pack provides; the site lists the substance under the name Myostatin, and the compound is YK-11. Neither clearance nor a urine window has been published for it, so any half-life figure circulating online is an extrapolation from other SARMs rather than a measurement. It does not aromatise.
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YK 11 SARM - Dragon Pharma
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$145.00
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Quality First

All products are manufactured under strict quality standards and independently tested before release. By purchasing, the buyer agrees to use these products in compliance with all applicable laws.

Product Overview

YK-11 is the odd one out in the SARM shelf: a molecule with a steroidal backbone rather than the non-steroidal frame that LGD-4033 or ostarine use, classed as a selective androgen receptor modulator because of how it behaves at the receptor rather than because of its chemistry. It acts as a partial agonist there, and laboratory work places its anabolic potency above DHT in cell models. The name attached to the card in this catalogue is Myostatin, which comes from the line of research the compound is best known for, and the pack itself is a single 10 mg tablet, 100 tablets to a box.

What the card cannot offer is a human profile. There is no published pharmacokinetics for YK-11, which means no elimination half-life, no clearance curve and no measured window for a drug test. The published work that exists is mechanistic and largely pre-clinical, so the material is presented here as a research compound rather than as something with an established course. Anyone comparing it with LGD 4033 or MK 2866 should note that both of those have trial data behind them and this one does not.

The one structural expectation that does carry over is a lack of aromatisation, so water retention and estrogenic effects are not part of the expected picture. The rest of the profile - suppression of the natural axis, liver load, how long any effect lasts - is reasoned from the SARM class rather than from YK-11 itself, and that distinction is worth keeping in view on every page that sells it.

Dosage Protocol

No human dose for this compound has been established, and extrapolating from cell-culture work is not a medical recommendation. The rows below therefore report the state of the sources rather than offering a schedule.

Beginner Not established: no human dose appears in the literature, and the 10 mg tablet is the only fixed unit the pack offers
Intermediate Not established: amounts used in the non-medical market are copied across from other SARMs rather than studied for this one
Advanced Not established: no upper range can be quoted, and no source describes a tolerance ceiling
Female No confirmed female protocol exists in the sources; the compound carries an androgenic profile, and nothing on the female side has been characterised
Administration Oral tablet; the pack is a single 10 mg strength, and the sources record no dosing interval to follow
Reference point Published research is in vitro and animal work on myostatin inhibition and AR signalling, not a human dose-finding study

Research Context and Laboratory Notes

The intellectual case for YK-11 rests on myostatin, a signalling protein that puts a brake on muscle growth. A 2021 paper describes the compound in that role, and the mechanism is genuinely interesting: block the brake and the same training stimulus may produce more tissue. In vitro work reports anabolic activity above DHT, while animal data raise a separate flag about oxidative stress and mitochondrial function. Both findings come from models, not from people.

That gap shapes how the material should be treated in a laboratory setting. Because no human clearance data exist, no washout period can be calculated and no accumulation profile can be predicted; sequential work with the same subject has no evidence base to stand on. Basic handling is the ordinary one for a sealed oral product: keep it in the original pack, dry and out of direct light, and respect the printed expiry date. The sources behind this card record no stability study for the tablet, so no shelf life beyond the manufacturer marking can be quoted.

Where a comparison is wanted inside the same catalogue section, GW501516 Cardarine is a different class of research material altogether, and MK 677 works through the growth hormone axis rather than the androgen receptor, so the three cannot be treated as interchangeable bench reagents.

What to Expect

  • Timeline. The only timeline available is the SARM pattern, two to four weeks before anything is noticeable; no measurement exists for this molecule specifically.
  • Strength and density. The reported picture is force output and a denser look without subcutaneous water, which follows from non-aromatisation rather than from a trial result.
  • Partial agonism. Because receptor activation is partial, the effect profile cannot be read across from a full androgen, even when the doses look comparable on paper.
  • Libido and drive. A falling libido is described as a class effect of suppressive SARMs, so it belongs in the expected list rather than the surprise list.
  • Honest limit. No human data cover dose, adverse events or pharmacokinetics for this compound, so every expectation here is class-level.
  • Regulatory limit. SARM products as a group have drawn FDA warnings, and user reports of liver injury are documented in review literature.
  • After stopping. Recovery has to be planned the way it is planned after any suppressive SARM, with a SERM and follow-up bloodwork.

Side Effects and Management

Nothing here is quantified for YK-11 itself, so each entry names the class risk and the monitoring route that would catch it early.

Liver injury, cholestatic or hepatocellularALT and AST before the run and again during it, a short course, and no second oral with liver load; described in SARM users as a class risk.
Suppressed natural testosteroneTestosterone with LH and FSH results, followed by SERM-based recovery; a class effect rather than a measured YK-11 finding.
Lipid shiftA full lipid panel, with attention to HDL; class risk, no compound-specific figures.
Irritability and poor sleepReduce the amount or stop; user reports only, never confirmed in a controlled setting.
Oxidative and mitochondrial stressObserved in an animal study and not transferable to a human plan; treat it as a reason for caution rather than a protocol.
No estrogenic effectsThe compound is not aromatised, so an aromatase inhibitor such as Arimidex does not belong in the plan and would only add its own risks.

Post-Cycle and Follow-Up

Recovery cannot be dated for this compound, because there is no clearance figure to count from. What can be described is the shape of the restart used across the SARM group in this catalogue, applied with that uncertainty stated openly.

OnsetNot established. Without a published half-life, the safest reading is to wait for clearance rather than to pick a fixed number of days
Option ANolvadex at 40 mg on the first fortnight, stepping down to 20 mg afterwards
Option BClomid at 50 mg, or Toremfine as the alternate SERM, run for four to six weeks
Mild suppressionA two to four week course is sometimes used when the axis is only lightly affected; for YK-11 that judgement has no data behind it and rests on bloodwork
Follow-upPanel of testosterone, LH/FSH, ALT/AST and a lipid profile, drawn after the SERM course rather than at the moment it ends
This material is published for educational and research reference purposes only. The compounds described are research-grade materials supplied for laboratory work and are not presented here as medicines or as a treatment for any condition. Dosing information reflects published clinical and reference data for the active substances, not a recommendation or a prescription. Nothing on this page should be read as medical advice. Keep all products out of reach of children and follow the regulations that apply in your country.
What is YK 11?
YK-11 is a steroidal selective androgen receptor modulator, sold here by Dragon Pharma as a 10 mg tablet, 100 to a box. The site files the substance under the name Myostatin because of the research line it is associated with. It has never been approved and it has no human pharmacokinetic profile, so it is a laboratory material rather than a medicine.
Is YK-11 a steroid or a SARM?
Both descriptions get used and the precise one is a SARM with a steroidal backbone: it is grouped with the receptor modulators because it acts as a partial agonist at the androgen receptor, yet its chemistry is not the non-steroidal scaffold that ostarine or LGD-4033 use. That mixed identity is exactly why its behaviour cannot be predicted from an anabolic steroid profile.
Is YK 11 a myostatin inhibitor?
That is the activity the compound is best known for in the literature: a 2021 paper examines it in the myostatin pathway, and the reasoning is that removing a natural brake on muscle growth lets training produce more tissue. The statement is a research claim from that paper, not a documented outcome in people, and no human trial has tested the myostatin route with this molecule.
YK 11 dosage - what do the sources establish?
Nothing. No human dose-finding work exists, and extrapolating from cell studies is not a medical recommendation. Figures quoted around the market are borrowed from other SARMs, and the only fixed number connected to the product is the 10 mg fill of the tablet. Where a dose cannot be sourced, the honest answer is that none can be given.
YK 11 side effects - what is known?
For this molecule specifically, nothing is quantified. The risks that can be named come from the SARM group: cholestatic and hepatocellular liver injury in user reviews, a downward shift in HDL, and suppression of the body's own testosterone. Irritability and disturbed sleep appear in user accounts but have never been studied. An animal study also reports oxidative and mitochondrial stress, which is a caution rather than a human finding.
Does YK-11 suppress testosterone?
The assumption across this class is that it does, because every SARM studied so far feeds back on the hypothalamus and pituitary to some degree. No measurement has been published for YK-11, so the amplitude and the duration are unknown. The practical consequence is that a post-run panel of testosterone with LH and FSH is worth taking, and that low drive in the weeks after a course should not be dismissed.
Do you need a PCT after YK-11?
If the axis was suppressed, the restart step looks the same as it does for the rest of the group: Nolvadex from 40 mg down to 20 mg, or Clomid at 50 mg for four to six weeks. The catch is timing: with no half-life on record, nobody can say how many days to wait, so clearance has to be assumed rather than calculated.
Is Dragon Pharma YK 11 legit, or are counterfeit batches around?
Because the molecule has no human data to check a result against, a wrong fill here is harder to notice than anywhere else, and brand threads about counterfeits exist for exactly this reason. What can be verified is the physical presentation: intact seal, printed batch and expiry, even tablet weight, and a vendor page that states the fill and how potency was tested.

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